Posts

Showing posts with the label MEDICINE AND HEALTH

ERECTILE DYSFUNCTION: CAUSES, COMPLICATION, PREVENTION AND TREATMENT

Image
❂✿❂✿❂✿❂✿❂ You might have been wondering why your penis doesn't stand for a long time before going back to its normal state. It must have been a thing of concern to you that you don't have good penile erection to satisfy your partner.😔 At times your partner may not complain and act like everything is normal. She just doesn't know how to tell you for fear of making you embarrassed. Without ejaculation, your penis is meant to remain standing (erected) upon sexual stimulation unless the arousal is suspended.  As far as there's sexual stimulation and without ejaculation, there should be erection.  ❂✿❂✿❂✿❂✿❂ For you to have erection, there must be increased blood flow to your penis. As this happens, your arteries and arterioles (Which are blood vessels that take blood from the heart to an organ) of your penis will dilate and your veins(which take blood away from an organ and back to the heart) will be compressed so that blood will no leave your penis. The build u

A NATURAL RESISTANCE TO HIV INFECTION.

Image
A NATURAL RESISTANCE TO HIV INFECTION. By Anyikwa Chukwuemeka Louis INTRODUCTION It is without doubt that the human immunodeficiency viral disease is the most widespread disease in recent human history. It is a plague, a menace and a nightmare. A cure has eluded us all for the better part of the time we have known this virus. HUMAN DEFICIENCY VIRUS The human immunodeficiency viruses (HIV) are two species of Lentivirus (a subgroup of retrovirus) that causes HIV infection and over time acquired immunodeficiency syndrome (AIDS). HIV infects vital cells in the human immune system, such as helper T cells (specifically CD4+ T cells), macrophages, and dendritic cells. HIV infection leads to low levels of CD4+ T cells through a number of mechanisms: 1. Pyroptosis of abortively infected T cells 2. Apoptosis of uninfected bystander cells 3. Direct viral killing of infected cells 4. Killing of infected CD4+ T cells by CD8+ cytotoxic lymphocytes that recognize infected cells. When CD4+